uillain-Barre syndrome complicating chronic active hepatitis is rare. In its antecedent
and associated illness, various viral infections are commonly involved (esp. viral hepatitis). A
classic features that allow diagnosis of GBS is ascending symmetrical weakness, areflexia,
minimal sensory loss, and albuminocytological dissociation in CSF.
A number of neurological complications of virl hepatitis have been reported, including
hepatic coma, hemorrhages into the CNS, both subdural and intraparenchymal, encephalitis,
meningitis, myelitis, isolated pyramidal tract and extrapyramidal tract dysfunction, Meniere's
disease, dermatomyositis, seizures, mononeuritis, plexitis, and psychiatric disturbances. But
there were no reports about GBS complicating chronic active viral hepatitis in Korea until
now.
We report a case of GBSS complicating chronic active hepatitis in a 41-year-old male.
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